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. 2020 May 18;10:8138. doi: 10.1038/s41598-020-65100-2

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Relative risk of all-cause and cardiovascular mortality as a function of HDL-C, apoA-I, HDL-P, SHDL-P and LHDL-P. Graphic represents hazard ratios (dots) and corresponding 95% confidence interval (95% CI) for risk of all-cause and cardiovascular mortality per 1 standard deviation increase of HDL-C, apoA-I, HDL-P, SHDL-P or LHDL-P. Among the 78 deceased patients analyzed, the number of CV death was 50. Model 1: adjusted for age, smoking, treatment for dyslipidemia, eGFR, LVEF, duration of CAD and Gensini score. Model 2: adjusted for treatment for dyslipidemia, LVEF, duration of CAD and Gensini score.