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. 2019 Jun 3;27(5):330–340. doi: 10.1038/s41417-019-0107-9

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — Pathway involved in the pathogenesis of BMP1 in TCGA-KIRC with GSEA. Enrichment curves are shown for activated gene sets related to a EMT, b hypoxia pathway, c angiogenesis, d G2M checkpoint, and e KRAS signaling. BMP bone morphogenetic protein, TCGA-KIRC The Cancer Genome Atlas kidney renal clear cell carcinoma, GSEA gene set enrichment analysis, EMT epithelial–mesenchymal transition, KRAS Kirsten rat sarcoma viral oncogene