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. 2020 May 22;11:2578. doi: 10.1038/s41467-020-16306-5

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — a–c The glucose consumption (a), lactate production (b), and ATP levels (c) in Mettl3^Mut/- HeLa, sh-Mettl3 Huh7 and their corresponding control cells. d, e The cellular ECAR (d) and OCR (e) were measured in wild type and Mettl3^Mut/- HeLa cells. f, g The cellular ECAR (f) and OCR (g) were measured in sh-control and sh-Mettl3 Huh7 cells. b The glucose consumption, lactate production, and ATP levels in Huh7 cells transfected with vector control or ALKBH5 constructs. Data are presented as the mean ± SD from three independent experiments. **p < 0.01 by two tailed t test for (a) (p = 0.005 and p < 0.0001), (b) (p < 0.0001), (c) (p < 0.0001), and (h) (p < 0.0001).