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. 2019 Dec 20;27(6):1998–2013. doi: 10.1038/s41418-019-0483-6

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© The Author(s), under exclusive licence to ADMC Associazione Differenziamento e Morte Cellulare 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — HeLa cells stably expressing pLKO.1-vector or pLKO.1-hnRNP F (#1 or #2) co-transfected with siNC, sihnRNP H1 or sihnRNP H1/H2, and then a colony formation assay was performed, b cell proliferation was determined by CCK-8 assay. c HeLa cells stably expressing pLKO.1-vector, or pLKO.1-hnRNP F (#1), or hnRNP F (#1) co-transfected with sihnRNP H1/H2, were subjected to wound healing assay. Scale bar, 0.1 mm. Data were quantified and graphed. Error bars represent means ± SD (n = 3). Statistical analysis was performed using Student's t test (***P < 0.001). HeLa cells as described in c were subjected to the transwell migration assay (d), transwell invasion assay (e), and cell cycle analysis (f). Scale bar, 10 μm. Error bars represent means ± SD (n = 3). Statistical analysis was performed using Student's t test (**P < 0.01, ***P < 0.001).