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. 2020 May 24;39:92. doi: 10.1186/s13046-020-01587-x

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — MSI2 knockdown promoted TNBC cell growth and migration in vitro.a Western blot and qRT-PCR assays. MSI2 expression was detected in multiple breast cancer cell lines. b Western blot and qRT-PCR assays. The efficiency of MSI2 knockdown in Hs-578T and BT20 cells. c CCK-8 assay for cell viability. Cell proliferation was assessed after MSI2 knockdown in Hs-578T and BT20 cells. d Wound-healing assay. Images of the wound-healing assay data showing the wound-healing ability in MSI2-knockdown Hs-578T and BT20 cells. Scale bar, 200 μm. e Transwell migration assay. Images showing the migration capacity in MSI2-knockdown Hs-578T and BT20 cells. Scale bar, 100 μm.