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. 2020 May 19;8(1):e000892. doi: 10.1136/jitc-2020-000892

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© Author(s) (or their employer(s)) 2020. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ.

This is an open access article distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited, appropriate credit is given, any changes made indicated, and the use is non-commercial. See http://creativecommons.org/licenses/by-nc/4.0/.

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Acute inflammation in COVID-19 and treatment strategies. In response to the viral infection, inflammatory monocyte and macrophage activation in the pulmonary luminal epithelium leads to a massive release of proinflammatory cytokines including IL-6, IL-1 and TNF-α (the different treatment strategy should be highlighted). These are potential targets currently under investigation. DAMPS, damage-associated molecular patterns; gp130, glycoprotein 130; IFN, interferon;IL, interleukin; JAK, Janus kinase; R, receptor; sIL-6R, soluble IL-6 receptor;TNF, tumor necrosis factor.