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. 2020 May 24;20:188. doi: 10.1186/s12935-020-01275-y

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — SALL4 knockdown inhibits glycolysis and suppresses gastric cancer growth and metastasis though the downregulation HK-2 in vivo. a, e Representative images of tumors from mice injected with control and SALL4 knockdown MGC-803 cells subcutaneously and intraperitoneally (n = 5 per group). b Representative images of HE staining and immunohistochemical staining of SALL4 and HK-2. c, d, f, g qRT-PCR and western blot analyses of glycolysis-related genes and proteins in tumors from mice injected with control and SALL4 knockdown MGC-803 cells. Vertical bars represented SD of the mean values (n = 3); *P < 0.05, **P < 0.01, ***P < 0.001. Scale bar: 50 μm