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. 2020 Mar 3;47(7):1757–1768. doi: 10.1007/s00259-020-04736-8

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — Acute anti-inflammatory effect of early enalapril therapy on heart after MI. a Representative polar maps of left ventricular ^99mTc-sestamibi perfusion (upper) and TSPO signal (middle) in sham and MI with and without early enalapril treatments. TSPO signal normalized to perfusion (both lower panels) shows elevated signal in the infarct territory of MI compared with sham, attenuated by enalapril. Semi-quantitative analysis of perfusion-normalized TSPO signal in b infarct territory and c remote myocardium. noTx, untreated; eACE_i, early enalapril