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. 2020 May 27;37(12):1452–1462. doi: 10.1089/neu.2016.4644

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© Zachary S. Bailey, et al., 2020; Published by Mary Ann Liebert, Inc.

This Open Access article is distributed under the terms of the Creative Commons Attribution Noncommercial License (http://creativecommons.org/licenses/by-nc/4.0/) which permits any noncommercial use, distribution, and reproduction in any medium, provided the original author(s) and the source are credited.

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FIG. 5. — Cerium oxide nanoparticles (CeONPs) decreased oxidative macromolecular damage after mild traumatic brain injury (mTBI) in vivo. Panel a demonstrates the decrease in lipid hydroperoxides formed after mTBI in CeONP-treated rats, with the chronic high dose group showing lipid hydroperoxides equal to those of sham animals. Panel b demonstrates a decrease in 3-nitrosotyrosine levels in animals treated with CeONP. Again, the chronic high dose reduced levels to that of sham animals.*Significant from sham, p < 0.01; #Significant from injury, p < 0.01.