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. 2020 May 25;20:469. doi: 10.1186/s12885-020-06890-6

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — Genes upregulated by androgen (R1881), hypoxia and HIF1a in LNCaP cells. a, 47 genes upregulated by androgen (LNCaP vehicle control vs. LNCaP R1881, right circle) were independently upregulated by hypoxia (LNCaP normoxia vs. LNCaP 1% hypoxia, left circle). b, 7 genes upregulated by HIF1a overexpression (LNCaP Empty vs. LNCaP HIF1a, left circle) were also independently upregulated by androgen (LNCaP Empty vehicle control vs. LNCaP Empty R1881, right circle). Three genes were independently upregulated by and androgen, hypoxia and HIF1a (SPRED1, NDRG1 and IGFBP)