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. 2020 Apr 8;44(1):185–195. doi: 10.3892/or.2020.7579

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Copyright: © Du et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 4. — AXL is highly expressed in drug-resistant cells. (A) RNA sequencing showed that AXL is a potential target of miR-625-3p. (B) AXL is overexpressed in gefitinib-resistant HCC827GR cells based on RNA sequencing data. (C) The GSE38121 dataset showed that AXL is highly upregulated in erlotinib-resistant HCC827ER cells. (D) The GSE71587 dataset showed higher expression of AXL in erlotinib-resistant HCC4006ER cells. ***P<0.001, compared to parental cell line. AXL, AXL receptor tyrosine kinase.