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. 2020 Mar 20;57(6):2521–2538. doi: 10.1007/s12035-020-01900-x

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — MT7 improves mitochondrial function in sensory neurons cultured from STZ-induced diabetic rats. a Oxygen consumption rate (OCR) was measured in the DRG from a 6-week-old diabetic rat cultured overnight and in the presence or absence of 100 nM MT7 for 1 h and in the presence or absence of a 3-h STO-609 (3 μM) pretreatment. OCR in pmol/min was normalized to mg of protein. b Maximal respiration and c spare respiratory capacity were determined after subtracting the non-mitochondrial OCR. Values are mean ± SEM, n = 6–7 replicate cultures, and adjusted to total protein levels. *p < 0.05 control vs MT-7 (one-way ANOVA with Tukey’s post hoc test)