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. 2020 Apr 29;44(1):139–155. doi: 10.3892/or.2020.7601

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Copyright: © Gao et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 2. — miR-193b inhibits cell cycle progression and induces apoptosis. (A) Transfection of F5M2 cells with miR-193b mimic induced cell cycle arrest in G₁ phase, (B) whereas transfection of F4 cells with miR-193b inhibitor resulted in an increased percentage of cells in S and G₂ phases. (C) Upregulation of miR-193b induced apoptosis of F5M2 cells, (D) whereas downregulation of miR-193b in F4 cells inhibited apoptosis. Data are presented as the mean ± SD of at least three independent experiments. *P<0.05. miR-193b, microRNA-193b; NC, negative control; PI, propidium iodide.

Figure 2. — miR-193b inhibits cell cycle progression and induces apoptosis. (A) Transfection of F5M2 cells with miR-193b mimic induced cell cycle arrest in G₁ phase, (B) whereas transfection of F4 cells with miR-193b inhibitor resulted in an increased percentage of cells in S and G₂ phases. (C) Upregulation of miR-193b induced apoptosis of F5M2 cells, (D) whereas downregulation of miR-193b in F4 cells inhibited apoptosis. Data are presented as the mean ± SD of at least three independent experiments. *P<0.05. miR-193b, microRNA-193b; NC, negative control; PI, propidium iodide.