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. 2020 May 29;22:126. doi: 10.1186/s13075-020-02221-z

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — Levels of circulating immune complexes are associated with autoantibodies and type I IFNs. a Levels of circulating ICs, analyzed by both IC-FLOW and commercial ELISA, were compared between time points of the absence or presence of anti-dsDNA antibodies. b Levels of circulating ICs were analyzed in patients with high disease activity and stratified based on the presence of anti-C1q antibodies (aC1q). c Levels of ICs were correlated with type I IFN activity score. Statistical analyses were performed using the Mann-Whitney U test and Spearman’s correlation with *p < 0.05,**p < 0.01, and ***p < 0.001