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. 2020 May 29;20:205. doi: 10.1186/s12935-020-01289-6

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — HMGB1 and GPX4/p-p65 correlation analysis in colon cancer tissues. We performed immunohistochemistry and analyzed the expression and correlation of HMGB1, GPX4 and p-p65 by tissue microarray containing 50 pairs of colon tumor tissues, and found that HMGB1, GPX4 and p-p65 were expressed in the tumor tissues (a) and correlation analysis showed that HMGB1 was positively correlated with GPX4 and p-p65 (b). c Western blot indicated acetylated HMGB1 and p-P65 expression were increased by using of cancer and adjacent tissue from seven patients with colon cancer. Scale bars: 50 μm