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. 2020 May 30;17:171. doi: 10.1186/s12974-020-01845-x

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Change of sensorimotor functions and brain infarct volume after ischemic stroke in rats. Neurological functional change after MCAO in rats as shown in Longa test (a). Sensorimotor functional change after MCAO in rats as evaluated by adhesive removal test (b, c), Limb-use asymmetry test (cylinder) (d) and modified grip-traction test (e). Change of brain infarct volume after MCAO was assessed by cresyl violet staining (f, g). n = 8–15, results are expressed as means ± SEM. ^###P < 0.001, ^##P < 0.01, ^#P < 0.05 versus the Sham group. ^***P < 0.001, ^**P < 0.01, ^*P < 0.05 versus the 4 days MCAO group