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. 2020 Jun 2;10:8998. doi: 10.1038/s41598-020-65763-x

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fundus autofluorescence images obtained from patients with mutations in phototransduction genes (PT). The short-wavelength autofluorescence (SW-AF) and near-infrared autofluorescence (NIR-AF) images obtained from patients with mutations in phototransduction genes (RHO, PRPF31, PDE6B, GUCA1A, TULP1, PDE6A, CNGB1, and GUCY2D), exhibit hyperautofluorescent rings in both SW-AF and NIR-AF images. The exception is the patient with a mutation in GUCY2D, who presents with a sectoral hyperautofluorescent arc in the superior retina. In all cases, autofluorescence was sufficient to enable imaging and the macula appeared brighter than the ophthalmic vessels and the optic nerve.