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. 2020 May 27;11:890. doi: 10.3389/fimmu.2020.00890

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Copyright © 2020 Xi, Chen, Yang, Zhang, Zhang, Guo, Zhao, Xue, Li and Zhang.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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MiR-128 inhibited the cell proliferation, migration, and invasion of PDAC cells. (A) Cell Counting Kit-8 (CCK-8) assay: the proliferation of miR-128 lentivirus-infected cells was lower than that of the control group at 24 and 48 h. (B) Clonogenicity assays: the miR-128 lentivirus-infected cells had a significantly lower survival rate than the control cells. (C,D) Wound healing assay: the migration of miR-128 lentivirus-infected cells was slower than that of the control cells. (E,F) Transwell assay: the percentage of cells that invaded through the Matrigel was lower in miR-128 lentivirus-infected cells than in the control cells. (G,H) The expression of genes associated with epithelial–mesenchymal transition (EMT) after mimic transfection and lentivirus infection. The results represent the mean ± SEM from three independent experiments. Comparisons between groups were determined by Student's t-test. ***p < 0.001.