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. 2020 May 27;11:890. doi: 10.3389/fimmu.2020.00890

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Copyright © 2020 Xi, Chen, Yang, Zhang, Zhang, Guo, Zhao, Xue, Li and Zhang.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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miR-128 promotes anti-tumor immunity through the ZEB1/CD47 axis. (A,B) The expression of CD47 and ZEB1 were downregulated in miR-128 mimic-transfected Panc02 cells. (C,D) The expression of CD47 and ZEB1 were downregulated in miR-128 lentivirus-infected Panc02 cells. (E,F) The expression of CD47 and ZEB1 were downregulated in miR-128 mimic-transfected H7 cells. (G,H) The expression of CD47 and ZEB1 were downregulated in miR-128 mimic-transfected PANC1 cells. (I,J) The expression of ZEB1, miR-128, and CD47 in ZEB1 knockdown Panc02 cells. (K) A schematic of the molecular mechanism by which miR-128 causes tumor regression. These results represent the mean ± SEM from three independent experiments. Comparisons between groups were determined by Student's t-test. *p < 0.05, **p < 0.01, and ***p < 0.001.