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. 2020 Jun 2;11(3):e01087-20. doi: 10.1128/mBio.01087-20

FIG 4.

FIG 4

Anti-integrin antibodies block activation of alveolar epithelial cell EGFR. Representative immunoblots (A) and densitometric analysis (B) show that R. delemar infection induced phosphorylation of EGFR on tyrosine residue 1068 compared to that in the control and that anti-integrin β1 antibody blocked it. Data in panel B are means ± standard deviations from three independent experiments.

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