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. 2020 Jun 1;20:164. doi: 10.1186/s12906-020-02968-7

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 10 — Comparison of protective effects of FA and OA on injured HUVECs. a Protein expressions of LC3II, p62, LOX-1, VCAM1, MCP-1, β-actin in HUVECs. The cells were treated with 40 mg/L ox-LDL (model). In addition, cells were pretreated with OA or FA for 2 h followed by 40 mg/L ox-LDL exposure for another 24 h. b, c, d, e, f Bar charts show the mean intensity of every protein quantified and normalized versus β-actin expression. Values are submitted as mean ± SD based on seperate experiments in triplicate.(*) P < 0.05, (**) P < 0.01 and (***) P < 0.001 versus model