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. 2020 May 14;42(1):463–473. doi: 10.1080/0886022X.2020.1762647

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© 2020 The Author(s). Published by Informa UK Limited, trading as Taylor & Francis Group.

This is an Open Access article distributed under the terms of the Creative Commons Attribution-NonCommercial License (http://creativecommons.org/licenses/by-nc/4.0/), which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 1. — Expression of miR-218 and its effect on cells proliferation after HG treatment. (a) Relative expression of miR-218 in DKD tissues and control based on GSE114477 dataset from public database. (b) Relative expression of miR-218 in human renal proximal tubule (HK-2) cells after HG treatment. **p < 0.01 vs. Con or NC. (c) CCK8 assays were used to detect the cells proliferation after HG-damaged with transfection of miR-218 mimic/inhibitor or NC. **p < 0.01. DKD: diabetic kidney disease; HG: high glucose; NC: negative control; Con: control.