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. 2020 May 5;39(23):4551–4566. doi: 10.1038/s41388-020-1312-6

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — a Gene expression clustering diagram between U251 cells (NC: control) and U251 cells that stably expressed LRRC4 (C4: LRRC4). b KEGG pathway enrichment using differentially expressed genes between U251 cells (NC: control) and U251 cells that stably expressed LRRC4 (C4: LRRC4). The gene ratio means the proportion of genes related to this signalling pathway in the differentially expressed genes. Correlation analysis of LRRC4 and autophagy-related genes MAP1LC3Band BECN1 in Cohort 1 (Glioblastoma, TCGA, Cell 2013) (c) and Cohort 2 (Glioblastoma Multiforme, TCGA, PanCancer Atlas) (d) of GBM samples from TCGA database. Kaplan–Meier curve depicting survival of patients with low and high LRRC4 (e), BECN1 (f) in TCGA.