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In addition to the independent pathway analyses, we had an a priori hypothesis that genes involved in interneuron development and migration would be altered in these groups. Specifically, those genes associated with MGE-derived interneurons were downregulated (Arx, Dlx1, Dlx5, Lhx6, & Nkx2.1) whereas markers of CGE-derived interneurons (Sox6, Gsx1, & Mash1) were not significantly affected.
