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. 2020 Jun 4;11:2817. doi: 10.1038/s41467-020-16309-2

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — a Kaplan–Meier tumor-free survival of AGO2^+/+;KRAS^G12D;Trp53^fl/+;p48Cre, AGO2^fl/+;KRAS^G12D;Trp53^fl/+;p48Cre, and AGO2^fl/fl;KRAS^G12D;Trp53^fl/+;p48Cre mice. b Chart showing PDAC (within the pancreas), the different metastatic lesions, and abnormal pathologies (black boxes) observed in each mouse of the indicated genotypes. c Representative H&E and AGO2 IHC in the indicated genotype. Scale bar, 100 µm.