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. 2020 Jun 5;20:218. doi: 10.1186/s12935-020-01314-8

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — Knockdown of CCT3 suppresses the proliferation of breast cancer cells, HCC1937 and MDA-MB-231 by MTT assay. MTT assay was carried out at 24 h, 48 h, 72 h, 96 h and 120 h after infection of shCCT3 and shCtrl. The number of cells was counted using a microplate reader at a wavelength of 490 nm, and the cell proliferation curve was plotted according to the value of OD490