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. 2020 Apr 9;91(6):612–621. doi: 10.1136/jnnp-2019-322493

Table 1.

Subject characteristics. The clinical phenotype of symptomatic mutation carriers was behavioural variant FTD (n=37), primary progressive aphasia (n=7), FTD with amyotrophic lateral sclerosis (ALS) (n=4), ALS without FTD (n=3), memory-predominant FTD (n=1), progressive supranuclear palsy (n=1) and dementia not otherwise specified (n=1). Continuous variables are reported as medians±IQR

Non-carriers Presymptomatic carriers Symptomatic carriers P value
N 70 106 54
Sex, male (%) 31 (44%) 47 (44%) 32 (59%) 0.157*
Age at CSF collection, years 47 (40–58) 45 (34–56) 63 (56–69) <0.001†
MMSE 30 (29–30) 30 (29–30) 26 (24–28) <0.001†
CDR plus FTD modules 0 (0–0) 0 (0–0) 9 (3–10) <0.001†
Disease duration, years 3 (2–6)
NPTX2, pg/mL 990 (597–1373) 1003 (624–1358) 643 (301–872) <0.001‡
Gene-specific information GRN C9orf72 MAPT GRN C9orf72 MAPT P value
N 47 42 17 15 31 8
Age at CSF collection, years 54 (41–58) 43 (32–53) 42 (34–46) 64 (61–69) 60 (55–70) 61 (53–64) <0.001†
NPTX2, pg/mL 1072 (661–1406) 901 (534–1387) 1079 (389–1263) 741 (385–870) 609 (305–884) 561 (233–861) <0.001‡
Age at symptom onset, years 63 (54–66) 55 (49–62) 55 (52–58) 0.109†
Disease duration, years 2 (1–4) 4 (2–8) 3 (1–8) 0.238†

2 test.

†Kruskall-Wallis tests.

‡ANCOVA with age as covariate.

ANCOVA, Analysis of covariance; CDR, Clinical Dementia Rating scale; CSF, cerebrospinal fluid; FTD, frontotemporal dementia; MMSE, Mini Mental State Examination; NPTX, neuronal pentraxin.