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. 2020 Jan 30;43(3):1054–1064. doi: 10.1007/s10753-020-01190-4

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — Glucose homeostasis. WT, wild-type mice; 4ko, Casp1/Casp11/NE/PR3 knockout mice. a Glucose blood levels during the insulin tolerance test (ITT). b Glucose blood levels during an oral glucose tolerance test. c Glucose concentration in plasma at the end of the diet intervention. d Insulin concentration in plasma at the end of the diet intervention. All samples were measured in a single replicate. Data is represented as mean ± SEM. **p < 0.01, effect of the diet; ##p < 0.01, effect of the genotype.