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. 2020 May 6;20(1):427–435. doi: 10.3892/etm.2020.8722

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Copyright: © Zhang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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(A) Volcano map of 15 hub genes in the protein-protein interaction network according to the highest degree. (B) Expression of various genes in the tumor samples and adjacent normal samples, including ASPM (P<0.0001), AURKA (P<0.0001), BUB1 (P<0.0001), BUB1B (P<0.0001), CCNA2 (P<0.0001), CCNB1 (P<0.0001), CCNB2 (P=0.1067), CDC20 (P<0.0001), CDK1 (P<0.0001), KIF11 (P<0.0001), MAD2L1 (P<0.0001), NCAPG (P<0.0001), NDC80 (P<0.0001), TOP2A (P<0.0001) and TTK (P<0.0001). CDK1, cyclin-dependent kinase 1; CCNA2, cyclin A2; MAD2L1, mitotic arrest deficient 2 like 1; TOP2A, DNA topoisomerase IIα; BUB1, budding uninhibited by benzimidazoles 1; TTK, TTK protein kinase; NCAPG, non-SMC condensin I complex subunit G; NDC80, NDC80 kinetochore complex component; AURKA, aurora kinase A; KIF11, kinesin family member 11; CDC2, cell division cycle 20; ASPM, abnormal spindle microtubule assembly; Adj, adjacent.