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. 2020 Jun;145(6):1606–1614.e4. doi: 10.1016/j.jaci.2020.02.026

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© 2020 MRC Laboratory of Molecular Biology. Published by Elsevier Inc. on behalf of the American Academy of Allergy, Asthma & Immunology.

This is an open access article under the CC BY license (http://creativecommons.org/licenses/by/4.0/).

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Fig 6 — IL-17 receptor B (IL-17rb) expression by ILC2s is required to drive chronic allergic skin inflammation. A-D, Representative hematoxylin and eosin staining (A [left]) and epidermal thickness measurement (A [right]); quantitation of percentage of CD4⁺ T cells (B); and mRNA levels of Il13 (C), Ccl17, and Ccl22 (D) in skin of Rora^Cre/CreIl17rb^flox/flox mice and Rora^Cre/Cre controls mice subjected to chronic epicutaneous sensitization with ovalbumin (OVA). Values in C and D are expressed relative to the mean of saline (SAL)-sensitized controls. Results in A-D are representative of 2 independent experiments with 4 or 5 mice per group. ∗P < .05; ∗∗ P < .005. N.D., Not detected.