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. 2020 Jun 9;16:186. doi: 10.1186/s12917-020-02415-2

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — Ultrastructural changes in the BAEC following exposure to LPS endotoxin of P. multocida B:2. a Nuclear changes exhibited by peripheral nuclear condensation (long arrows) with mitochondrial swelling (short arrow) and dilated, fragmentation and vesiculation of RER membrane (arrowheads) (bar = 1 μm, TEM). b Blebbing of cytoplasmic membrane (long arrows), and swelling and cristolysis in mitochondria (short arrows) (bar = 500 nm, TEM). c Dilatation (long arrows) and vesiculation (short arrow) of RER. Ribosomal degranulation from RER resulted in ribosomal fragments in the cytosol. Note the discontinued nuclear membrane (arrowhead). (bar = 200 nm, TEM). d Discontinuation of plasma membrane (long arrow). Noted the RER vesiculation (short arrows) and swelling and cristolysis of mitochondria (arrowheads) and (bar = 2 μm, TEM)