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. 2020 Jun 9;21:219. doi: 10.1186/s12882-020-01880-z

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Representative immunohistochemical findings of Ki67 and hematoxylin counterstain (× 100) (a), and the differences in the mean labeling index of Ki67 protein expression (b) among the four groups: Control NP rats, Control HP rats, CKD NP rats, and CKD HP rats. a Nuclear Ki67 expression. Many nuclei in CKD HP were positively stained for the Ki67 protein. b In CKD HP rats, Ki67 expression was significantly increased compared with that of the other groups, then the number of Ki67 positive cells tended to increase in the Control NP rats and CKD NP rats, though not significantly different. Number of animals: Control NP, 6; Control HP, 6; CKD NP, 6; and CKD HP, 6. Results are presented as the mean ± SD. The mean difference is significant at the 0.0083 level for Bonferroni test