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. 2020 May 1;10(5):271. doi: 10.3390/brainsci10050271

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© 2020 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/).

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Preadministration of SR or genetic deletion of CB1Rs mitigates high-dose ethanol-induced loss of cytosolic ABC in the neonatal mouse brain. Western blot analysis of the ABC proteins in the HP and NC cytosolic extracts obtained 8 h after saline or ethanol treatment from different group (S + V, E + V, S + SR and E + SR; S + CB1RWT, E + CB1RWT, S + CB1RKO, and E + CB1RKO). The protein samples were equally loaded, confirmed with Ponceau S staining, and normalized to β-actin. Error bars, SEM (* p < 0.05 vs. S + V or S + CB1RWT group; # p < 0.05 vs. E + V or E + CB1RWT group, n = 8 pups/group).