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. 2020 Jun 10;20:225. doi: 10.1186/s12935-020-01288-7

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Significantly decreased ARHGAP30 expression in tumor tissues from patients with pancreatic cancer and pancreatic cancer cell lines. a Thirty paired cancer and adjacent tissues were collected from patients, and ARHGAP30 mRNA expression was detected using RT-PCR. b Statistical analysis of ARHGAP30 protein expression in 90 paraffin-embedded pancreatic cancer and adjacent tissues. c Immunohistochemical detection of ARHGAP30 expression and Kaplan–Meier survival analysis and log-rank comparison of 90 patients with pancreatic cancer, including 59 cases of death (high ARHGAP30 expression: 20, low ARHGAP30 expression: 39). d ARHGAP30 mRNA and protein expression in pancreatic cancer cell lines (ASPC1, BXPC3, MiaPaca2, PANC1 and SW1990) and normal human pancreas cells (HPC-Y5) was detected using RT-PCR and western blotting, respectively. *p < 0.05, **p < 0.01, ***p < 0.001 vs. HPC-Y5