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. 2020 Jan 22;221(12):1962–1972. doi: 10.1093/infdis/jiaa026

Table 5.

Prevalence of Nonpolymorphic and Polymorphic HIV-2 Integrase Mutations in INSTI-Naive and INSTI-Treated Persons Significantly Associated With INSTI Therapy

Mutation INSTI Naive, No. (%) (n = 236)a INSTI Treated, No. (%) (n = 60)a Fold Fisher Exact Test Pb
Nonpolymorphic
 Q91R 1 (0.42) 8 (13.33) 31.5 .0005
 E92A 0 (0) 5 (8.33) >>> .01
 E92Q 0 (0) 12 (20.00) >>> <.000001
 T97A 0 (0) 19 (31.67) >>> <.000001
 G140S 0 (0) 7 (11.67) >>> .0004
 Y143G 0 (0) 4 (6.67) >>> .05
 Q148R 0 (0) 7 (11.67) >>> .0004
 A153G 1 (0.42) 10 (16.67) 39.3 .00003
 N155H 1 (0.42) 17 (28.33) 66.9 <.000001
 H156R 2 (0.85) 10 (16.67) 19.7 .0001
 R231 insertion 0 (0) 7 (12.28) >>> .0003
Polymorphic
 I84V 15 (6.36) 19 (31.67) 5.0 .00004
 A119T 4 (1.69) 8 (13.33) 7.9 .02
 V141I 3 (1.27) 8 (13.33) 10.5 .008

Abbreviation: INSTI, integrase strand transfer inhibitor.

an = total number of persons from whom sequences were obtained. However, sequences encompassing position 231 were available from 57 (not 60) INSTI-treated persons.

bAdjustment for multiple hypothesis testing for all mutations occurring in ≥3 times in INSTI-experienced persons and ≥2 times more commonly in INSTI-experienced compared with INSTI-naive persons was performed using the Holm method.