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. 2020 Jun 5;11:736. doi: 10.3389/fpls.2020.00736

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Copyright © 2020 Katyayini, Rinne, Tarkowská, Strnad and van der Schoot.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Model of axillary bud (AXB) quiescence and activation. During their formation, AXBs accumulate GA_3/6, which upregulates GA2ox gene expression. GA2ox deactivates GA_1/4, thereby depriving the AXBs of GA_1/4-mediated signaling. Decapitation activates quiescent AXBs by rapidly reducing GA_3/6 levels and downregulating GA2ox transcription, thereby elevating GA_1/4 levels and signaling. Subsequently, the GA_1/4 biosynthesis gene GA3ox2 is upregulated dramatically increasing GA levels and promoting AXB outgrowth. Red arrows and text indicate AXB inhibitory effects. Green arrows and text indicate AXB activating and growth promoting effects.