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. 2020 Jun 5;11:418. doi: 10.3389/fneur.2020.00418

Figure 3.

Figure 3

18F-FDG-PET hypermetabolism in LGI1 AE patients with FBDS. (A) Neuroimaging comparison of the subgroups of LGI1 AE. (B) Representative 18F-FDG-PET hypermetabolism among different subgroups after SPM (P < 0.01) analysis. (C) The comparison of the 18F-FDG-PET hypermetabolic pattern between FBDS and non-FBDS. In the FBDS group, the abnormal PET signal more often appeared in the BG-only group and the BG + MTL group. However, in the non-FBDS group, the abnormal PET signal significantly appeared in the BG + MTL group. (D) A comparison of the 18F-FDG-PET hypermetabolic pattern in FBDS and non-FBDS. In the BG-only group, the frequency of FBDS was higher than that of non-FBDS (P < 0.001). LGI1, leucine-rich glioma-inactivated 1; AE, autoimmune encephalitis; FBDS, faciobrachial dystonic seizures; MRI, magnetic resonance imaging; PET, 18F-fluoro-2-deoxy-d-glucose positron emission tomography; MTL, medial temporal lobe; BG, basal ganglia. +, positive; –, negative.