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. 2020 Jun 5;11:418. doi: 10.3389/fneur.2020.00418

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Copyright © 2020 Liu, Shan, Zhao, Ren, Ren, Chen, Shi, Lv, Li, Liu, Ai and Wang.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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¹⁸F-FDG-PET characteristics of LGI1 AE patients in different clinical phases. FDG-PET hypermetabolism was specifically located in the BG and MTL, whether it was in the acute phase or chronic phase, and there is no statistical metabolic change between acute and chronic phases for BG and MTL. LGI1, leucine-rich glioma-inactivated 1; AE, autoimmune encephalitis; ¹⁸F-FDG-PET, ¹⁸F-fluoro-2-deoxy-d-glucose positron emission tomography; BG, basal ganglia; MTL, medial temporal lobe.