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. 2020 Jun 12;17:185. doi: 10.1186/s12974-020-01857-7

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 4 — CX3CR1 inhibits the Akt-nNOS pathway in the NTS of fructose-fed hypertensive rats. a Semi-quantitative immunoblot analysis determined the phosphorylation level of ERK1/2, Akt, nNOS, and eNOS protein in the NTS of fructose-fed rats. b The bar graph shows phosphorylated ERK1/2^T202/Y204, Akt^S473, nNOS^S1416_, and eNOS^S1177 in the NTS of hypertensive rats. One-way ANOVA with Scheffe’s post-hoc was performed for statistical analysis. The values are represented as mean ± SEM. *P < 0.05 compared to the control group. ^#P < 0.05 compared to fructose-fed rats(n = 6~8 per group)