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. 2020 Jun 12;20:234. doi: 10.1186/s12935-020-01311-x

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 5 — Knockdown of UBE2I inhibited invasion and migration of HCC cells. a The knockdown effect of si-UBE2I in HepG2 cells. b The knockdown effect of si-UBE2I in Bel7402 cells. c Treatment of si-UBE2I significantly suppressed HepG2 invasion. d The quantitative analysis of the effect of si-UBE2I on HepG2 invasion. e Treatment of si-UBE2I significantly suppressed Bel7402 invasion. f The quantitative analysis of the effect of si-UBE2I on Bel7402 invasion. g Treatment of si-UBE2I significantly suppressed HepG2 migration. h Treatment of si-UBE2I significantly suppressed Bel7402 migration. Bar scale: 100 um. P-value < 0.05 was considered as statistically significant