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. 2020 Jun 12;10:9591. doi: 10.1038/s41598-020-66858-1

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Schematic drawing of hypothetical mechanism of role of vascular running pattern in CSC development. In eyes with vessels of Haller’s layer running symmetrically, the watershed zone exists beneath the subfoveal region. Even when these vessels are dilated, the compression of the choroidal inner layer does not occur. While, in eyes with no symmetry, the watershed zone does not exist beneath the fovea. If the vessels are dilated, dilated choroidal vessels are likely to compress inner choroid, resulting in damage to foveal area. Thus, CSC may be developed.