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. 2020 Jun 12;48(6):0300060520922339. doi: 10.1177/0300060520922339

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© The Author(s) 2020

Creative Commons Non Commercial CC BY-NC: This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (https://creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 3. — The lncRNA SNHG12 promoted the development of EMT to strengthen the migration and invasion of HTR-8/SVneo cells. (A) Expression of EMT-related proteins (E-cadherin, β-catenin, and vimentin) was detected by western blotting after the overexpression of lncRNA SNHG12. (B) Levels of E-cadherin, β-catenin, and vimentin were detected by western blotting after knockdown of lncRNA SNHG12. *P < 0.05, **P < 0.01, ***P < 0.001. lncRNA, long noncoding RNA; SNHG12, small nucleolar RNA host gene 2; EMT, epithelial–mesenchymal transition.