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. 2020 Jan 14;40(6):939–954. doi: 10.1007/s10571-019-00784-3

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — Pallidal inflammation following STN–DBS in vivo. a–d Photomicrographs of immunoreactivity for GFAP in saline (a), 6-OHDA (b), 6-OHDA + DBS OFF, (c) and 6-OHDA + DBS ON (d) and e quantification of GFAP + cells in the GP (n = 4). f–i In vivo evaluation of expression levels of IL-1β (f), IL-6 (g), IL-10, (h) and IFN-γ (i) in the GP. Values represent the mean + SEM of four independent experiments. *p < 0.05; **p < 0.001, ***p < 0.0001 vs. saline-injected control animals. $$p < 0.001 vs. 6-OHDA group