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. 2020 Jun 15;10:113. doi: 10.1186/s13568-020-01033-3

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — The experimental animals were induced with spinal cord injury and were exposed to miR-466c-3p mimics or agomir-negative control followed by western blot analysis for determining the expression of cleaved-caspse-9, cleaved-caspase-3, pro-cspase-3, Bcl-2 and Bax in the spinal cord tissue subjected to injury, actin was selected as loading control. The results are mean ± SD. **P < 0.01 compared to spinal cord injury rats + treated with agomir-negative control; #P < 0.05, ##P < 0.01 compared to sham operated rats