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. 2020 Jun 17;11(6):467. doi: 10.1038/s41419-020-2673-z

Fig. 8. Delayed administration of 3-MA blocks inflammation responses in the kidney of hyperuricemic rats.

Fig. 8

The kidney tissue lysates were subjected to immunoblot analysis with specific antibodies against p-STAT3, STAT3, p-NF-κB, NF-κB, and GAPDH a. Expression level of p-STAT3 was quantified by densitometry and normalized with STAT3 b. Expression level of p-NF-κB was quantified by densitometry and normalized with NF-κB c. The kidney tissue lysates were subjected to immunoblot analysis with specific antibodies against CD68 and GAPDH d. Expression level of CD68 was quantified by densitometry and normalized with GAPDH e. Photomicrographs illustrating immunohistochemistry staining of MCP1, CD3, CD68 f. Data are represented as the mean ± SEM (n = 6). Means with different superscript letters are significantly different from one another (P < 0.05). All scale bars = 20 μm.