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. 2016 Jun 11;75(8):770–778. doi: 10.1093/jnen/nlw049

FIGURE 1.

FIGURE 1

Experimental traumatic brain injury (TBI) causes a biphasic response in apolipoprotein E (apoE) protein levels. (A) Soluble apoE protein levels from the ipsilateral cortex of sham and TBI-injured C57BL/6 mice. Representative blots are shown. (B) Comparison of diethylamine-soluble apoE (DEA) and radioimmunoprecipitation assay-soluble apoE (RIPA) from ipsilateral cortex of C57BL/6 mice. (C)Apoe mRNA quantified by RT-PCR from the ipsilateral cortex of a separate cohort of C57BL/6 mice. One-way ANOVA with Dunnett post hoc analysis comparing 1, 3, and 7 days to sham for all panels, except 1-way ANOVA with Tukey post hoc analysis for panel B ). *p < 0.05; **p < 0.01; ***p < 0.001.