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. 2020 Apr 19;39(25):4781–4797. doi: 10.1038/s41388-020-1284-6

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — a siKinome identified targets involved in palbociclib resistance (PalboR) (n = 2 biological and n = 3 technical replicates). b Effect of escalating doses of pictilisib (PI3K inhibitor), everolimus (mTORC1 inhibitor), AZD1775 (WEE1 inhibitor) and THZ1 (CDK7 inhibitor) in MCF7^PalboR, MCF7 LTED^PalboR, T47D^PalboR and T47D LTED^PalboR versus their corresponding parental cell lines (n = 3 biological and n = 8 technical replicates). Data represents % viable cells compared with vehicle control for each cell line. Error bars represent means ± SEM.