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. 2020 Mar 4;161(7):1670–1681. doi: 10.1097/j.pain.0000000000001858

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Copyright © 2020 The Author(s). Published by Wolters Kluwer Health, Inc. on behalf of the International Association for the Study of Pain.

This is an open access article distributed under the terms of the Creative Commons Attribution-Non Commercial-No Derivatives License 4.0 (CCBY-NC-ND), where it is permissible to download and share the work provided it is properly cited. The work cannot be changed in any way or used commercially without permission from the journal.

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Figure 1. — Timeline of the electrophysiological in vivo experiments. After the surgical procedure and the recording of baseline activity, either Ab181 (10 mg/kg) or vehicle (A5Su) was injected intravenously. After a resting period of 2.5 hours to allow for the distribution of the antibody throughout the circulation and the relevant tissues, sumatriptan or its vehicle was administered intravenously. Following another resting period of 30 minutes to allow sumatriptan to act, stimulus-evoked and spontaneous neuronal activity was recorded in the TCC. TCC, trigeminocervical complex.