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. 2020 Jun 18;11(6):472. doi: 10.1038/s41419-020-2581-2

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — a Schematic depicting key lncRNAs and hub genes in the LRSP subpathways and network. b Topology features of the key lncRNAs. c DLEU2 and its potential target genes (PIK3CD, PPP1CC, and PPP3R1) were significantly upregulated in the tumor tissues compared with the normal control tissues in HNSCC patients from the TCGA database. d The mRNA expression of DLEU2 was evaluated by qRT-PCR analysis in 66 cases of LSCC specimens and the matched adjacent normal tissues. e–h Correlations of DLEU2 expression with the clinicopathological characteristics of the tumor stage (e), lymph node metastasis (f), tumor node metastasis (g), and differentiation (h) in LSCC specimens. i The correlation between survival time and DLEU2 expression was analyzed in LSCC patients. **P < 0.01, ***P < 0.001.