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. 2020 Jun 18;11(6):472. doi: 10.1038/s41419-020-2581-2

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — a QRT-PCR analysis of DLEU2, PIK3CD, CCND1, CCNE1, MMP2, and MMP7 in the indicated cells. b Western blot analysis of PIK3CD, Akt, p-Akt, CCND1, CCNE1, MMP2, and MMP7 in the indicated cells. c Immunohistochemical scores for p-AKT, p-mTOR, CCND1, CCNE1, MMP2, and MMP7 in LSCC specimens, stratified by DLEU2 expression. d Immunohistochemical scores for PIK3CD, p-AKT, p-mTOR, CCND1, CCNE1, MMP2, and MMP7 in samples from the xenograft tumors. These results are presented as the mean ± SD from three independent experiments. *P < 0.05, **P < 0.01, ***P < 0.001.